INCREASED MATERNAL NON-OXIDATIVE ENERGY METABOLISM MEDIATES ASSOCIATION BETWEEN PRENATAL DI-(2-ETHYLHEXYL) PHTHALATE (DEHP) EXPOSURE AND OFFSPRING AUTISM SPECTRUM DISORDER SYMPTOMS IN EARLY LIFE: A BIRTH COHORT STUDY

Increased maternal non-oxidative energy metabolism mediates association between prenatal di-(2-ethylhexyl) phthalate (DEHP) exposure and offspring autism spectrum disorder symptoms in early life: A birth cohort study

Prenatal phthalate shades eq 07m exposure has previously been linked to the development of autism spectrum disorder (ASD).However, the underlying biological mechanisms remain unclear.We investigated whether maternal and child central carbon metabolism is involved as part of the Barwon Infant Study (BIS), a population-based birth cohort of 1,074 Aus

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ST-2191, an Anellated Bismorpholino Derivative of Oxy-Fingolimod, Shows Selective S1P1 Agonist and Functional Antagonist Potency In Vitro and In Vivo

Sphingosine 1-phosphate (S1P) is an thumper v2 price extensively studied signaling molecule that contributes to cell proliferation, survival, migration and other functions through binding to specific S1P receptors.The cycle of S1P1 internalization upon S1P binding and recycling to the cell surface when local S1P concentrations are low drives T cell

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